Tuesday, June 12, 2018

Immunoreactivity for the NMDA NR1 subunit in bulbospinal catecholamine and serotonin neurons of rat ventral medulla

Ida Llewellyn-Smith and Patrick J. Mueller
Autonomic Neuroscience: Basic and Clinical (2013)


Two of the major autonomic cell groups in the ventral medulla are the bulbospinal catecholamine- and serotonin-containing neurons. The number and distribution of glutamate receptors located on these two types of neurons is likely to shape their function and how they contribute to various physiological and pathophysiological processes. NMDA receptors are a type of glutamate receptor that are made up of a five subunits (pentameric), one of which, that will serve as a marker for NMDA receptors in this study, is the NR1 subunit. The purpose of this study was to determine the number of bulbospinal catecholamine- and serotonin-containing neurons that contained the NR1 subunit, and thus NMDA receptors. 
To test this hypothesis, a group of 10 rats were housed in the lab with unlimited food and water. The rat spinal cords were injected with a retrograde tracer called cholera toxin B (CTB) before perfusion and subsequent sacrifice. The rat brains were sectioned and triple immunofluorescently labeled for NR1 (NMDA marker), CTB (bulbospinal indicator), and either TH or TpTH (catecholamine and serotonin enzyme, respectively) on 12 sections in the RVLM. The reason this study focused on TH and TpTH neurons is because they are known to be important for sympathetic control of the cardiovascular system.  The stained sections were examined under a microscope and the NR1-positive neurons were counted.
The results showed that virtually all bulbospinal TH and non-TH neurons in the RVLM, and all bulbospinal TpTH and non-TpTH in raphe pallidus and parapyramidal region, express NR1. However, even though the NR1 subunit is considered obligatory for functional NMDA channels, it is possible that some of the neurons express the NR1 subunit but do not have functional NMDA channels. It is thought that NMDA-mediated neurotransmission in the RVLM may contribute to neuroplasticity.

- Ben Huber



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